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Being Open to the Unexpected. Mol Biol Cell.
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2019. Insulin/insulin-like growth factor signaling in C. elegans.. WormBook. :1-43.
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2013. Dictyostelium myosin 25-50K loop substitutions specifically affect ADP release rates.. Biochemistry. 37(19):6738-44.
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1998. The sequence of the myosin 50-20K loop affects Myosin's affinity for actin throughout the actin-myosin ATPase cycle and its maximum ATPase activity.. Biochemistry. 38(12):3785-92.
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1999. Variable surface loops and myosin activity: accessories to a motor.. J Muscle Res Cell Motil. 21(2):139-51.
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2000. A myosin II mutation uncouples ATPase activity from motility and shortens step size.. Nat Cell Biol. 3(3):311-5.
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2001. Genes that act downstream of DAF-16 to influence the lifespan of Caenorhabditis elegans.. Nature. 424(6946):277-83.
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2003. Using whole-genome transcriptional analyses to identify molecular mechanisms of aging. Drug Discovery Today: Disease Mechanisms. 3:41-46.
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2006. The search for DAF-16/FOXO transcriptional targets: approaches and discoveries.. Exp Gerontol. 41(10):910-21.
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2006. Aging: miRacles of longevity? Curr Biol. 20(24):R1076-8.
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2010. Tissue entrainment by feedback regulation of insulin gene expression in the endoderm of Caenorhabditis elegans.. Proc Natl Acad Sci U S A. 104(48):19046-50.
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2007. 9c. Short and sweet. Elife.
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2021. 4. Metformin rescues Parkinson’s disease phenotypes caused by hyperactive mitochondria. Proc Natl Acad Sci U S A..
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2020. 1. “Horizontal and vertical transmission of transgenerational memories via the Cer1 transposon. bioRxiv.
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2020. 7. Protocol for transgenerational learned pathogen avoidance behavior assays in Caenorhabditis elegans. STAR protocols.
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2018. Piwi/PRG-1 Argonaute and TGF-β Mediate Transgenerational Learned Pathogenic Avoidance. Cell. 177(7):-1841.e12.
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2019. Comparing genomic expression patterns across species identifies shared transcriptional profile in aging.. Nat Genet. 36(2):197-204.
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2016. The nematode Caenorhabditis elegans as a model for aging research. Drug Discov Today: Disease Models .
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